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Despite Increased Type 1 IFN, Autoimmune Nonobese Diabetic Mice Display Impaired Dendritic Cell Response to CpG and Decreased Nuclear Localization of IFN-Activated STAT1

Innate immune signals help break self-tolerance to initiate autoimmune diseases such as type 1 diabetes, but innate contributions to subsequent regulation of disease progression are less clear. Most studies have measured in vitro innate responses of GM-CSF dendritic cells (DCs) that are functionally...

תיאור מלא

שמור ב:
מידע ביבליוגרפי
הוצא לאור ב:J Immunol
Main Authors: Rahman, M. Jubayer, Rahir, Gwendoline, Dong, Matthew B., Zhao, Yongge, Rodrigues, Kameron B., Hotta-Iwamura, Chie, Chen, Ye, Guerrero, Alan, Tarbell, Kristin V.
פורמט: Artigo
שפה:Inglês
יצא לאור: 2016
נושאים:
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC5074843/
https://ncbi.nlm.nih.gov/pubmed/26826238
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1501239
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