Acute angiotensin-converting enzyme inhibition increases the plasma level of the natural stem cell regulator N-acetyl-seryl-aspartyl-lysyl-proline.

Angiotensin I-converting enzyme (ACE) has two homologous active NH2- and COOH-terminal domains and displays activity toward a broad range of substrates. The tetrapeptide N-acetyl-seryl-aspartyl-lysyl-proline (Ac-SDKP) has been shown to be hydrolyzed in vitro by ACE and to be a preferential substrate...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Päätekijät: Azizi, M, Rousseau, A, Ezan, E, Guyene, T T, Michelet, S, Grognet, J M, Lenfant, M, Corvol, P, Ménard, J
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: 1996
Aiheet:
Research Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC507123/
https://ncbi.nlm.nih.gov/pubmed/8609242
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