Epithelial-macrophage interactions determine pulmonary fibrosis susceptibility in Hermansky-Pudlak syndrome

Alveolar epithelial cell (AEC) dysfunction underlies the pathogenesis of pulmonary fibrosis in Hermansky-Pudlak syndrome (HPS) and other genetic syndromes associated with interstitial lung disease; however, mechanisms linking AEC dysfunction and fibrotic remodeling are incompletely understood. Since...

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Опубликовано в: :JCI Insight
Главные авторы: Young, Lisa R., Gulleman, Peter M., Short, Chelsi W., Tanjore, Harikrishna, Sherrill, Taylor, Qi, Aidong, McBride, Andrew P., Zaynagetdinov, Rinat, Benjamin, John T., Lawson, William E., Novitskiy, Sergey V., Blackwell, Timothy S.
Формат: Artigo
Язык:Inglês
Опубликовано: American Society for Clinical Investigation 2016
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Research Article
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC5070955/
https://ncbi.nlm.nih.gov/pubmed/27777976
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/jci.insight.88947
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