FAK Kinase Activity Is Required for the Progression of c-MET/β-Catenin-Driven Hepataocellular Carcinoma

There is an urgent need to develop new and more effective therapeutic strategies and agents to treat hepatocellular carcinoma (HCC). We have recently found that deletion of Fak in hepatocytes before tumors form inhibits tumor development and prolongs survival of animals in a c-MET (MET)/β-catenin (C...

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Bibliografiset tiedot
Julkaisussa:Gene Expr
Päätekijät: Shang, Na, Arteaga, Maribel, Zaidi, Ali, Cotler, Scott J., Breslin, Peter, Ding, Xianzhong, Kuo, Paul, Nishimura, Michael, Zhang, Jiwang, Qiu, Wei
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Cognizant Communication Corporation 2016
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Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC5064945/
https://ncbi.nlm.nih.gov/pubmed/27142958
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3727/105221616X691604
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