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Severe active C3 glomerulonephritis triggered by immune complexes and inactivated after eculizumab therapy
BACKGROUND: Understanding the role of alternative complement pathway dysregulation in membranoproliferative glomerulonephritis (MPGN) has led to a dramatic shift in its classification into two subgroups: immune complex-mediated MPGN and complement-mediated MPGN, consisting of dense deposit disease a...
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| Veröffentlicht in: | Diagn Pathol |
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| Hauptverfasser: | , , , , |
| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
BioMed Central
2016
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5055692/ https://ncbi.nlm.nih.gov/pubmed/27717365 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s13000-016-0547-6 |
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