ANGPTL2 activity in cardiac pathologies accelerates heart failure by perturbing cardiac function and energy metabolism

A cardioprotective response that alters ventricular contractility or promotes cardiomyocyte enlargement occurs with increased workload in conditions such as hypertension. When that response is excessive, pathological cardiac remodelling occurs, which can progress to heart failure, a leading cause of...

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Bibliografiske detaljer
Udgivet i:Nat Commun
Main Authors: Tian, Zhe, Miyata, Keishi, Kadomatsu, Tsuyoshi, Horiguchi, Haruki, Fukushima, Hiroyuki, Tohyama, Shugo, Ujihara, Yoshihiro, Okumura, Takahiro, Yamaguchi, Satoshi, Zhao, Jiabin, Endo, Motoyoshi, Morinaga, Jun, Sato, Michio, Sugizaki, Taichi, Zhu, Shunshun, Terada, Kazutoyo, Sakaguchi, Hisashi, Komohara, Yoshihiro, Takeya, Motohiro, Takeda, Naoki, Araki, Kimi, Manabe, Ichiro, Fukuda, Keiichi, Otsu, Kinya, Wada, Jun, Murohara, Toyoaki, Mohri, Satoshi, Yamashita, Jun K., Sano, Motoaki, Oike, Yuichi
Format: Artigo
Sprog:Inglês
Udgivet: Nature Publishing Group 2016
Fag:
Article
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC5052800/
https://ncbi.nlm.nih.gov/pubmed/27677409
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/ncomms13016
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