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Characterization of metabolic health in mouse models of fibrillin-1 perturbation
Mutations in the microfibrillar protein fibrillin-1 or the absence of its binding partner microfibril-associated glycoprotein (MAGP1) lead to increased TGFβ signaling due to an inability to sequester latent or active forms of TGFβ, respectively. Mouse models of excess TGFβ signaling display increase...
Tallennettuna:
| Julkaisussa: | Matrix Biol |
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| Päätekijät: | , , , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
2016
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4992667/ https://ncbi.nlm.nih.gov/pubmed/26902431 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.matbio.2016.02.006 |
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