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Characterization of metabolic health in mouse models of fibrillin-1 perturbation

Mutations in the microfibrillar protein fibrillin-1 or the absence of its binding partner microfibril-associated glycoprotein (MAGP1) lead to increased TGFβ signaling due to an inability to sequester latent or active forms of TGFβ, respectively. Mouse models of excess TGFβ signaling display increase...

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Bibliografische gegevens
Gepubliceerd in:Matrix Biol
Hoofdauteurs: Walji, Tezin A., Turecamo, Sarah E., DeMarsilis, Antea J., Sakai, Lynn Y., Mecham, Robert P., Craft, Clarissa S.
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: 2016
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Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4992667/
https://ncbi.nlm.nih.gov/pubmed/26902431
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.matbio.2016.02.006
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