Complement Destabilizes Cardiomyocyte Function in Vivo after Polymicrobial Sepsis and In Vitro ()

There is accumulating evidence during sepsis that cardiomyocyte (CM) homeostasis is compromised, resulting in cardiac dysfunction. An important role for complement in these outcomes is now demonstrated. Addition of C5a to electrically-paced CMs caused prolonged elevations of [Ca(2+)]i during diastol...

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Detalhes bibliográficos
Publicado no:J Immunol
Main Authors: Kalbitz, Miriam, Fattahi, Fatemeh, Herron, Todd J., Grailer, Jamison J., Jajou, Lawrence, Lu, Hope, Huber-Lang, Markus, Zetoune, Firas S., Sarma, J. Vidya, Day, Sharlene M., Russell, Mark W., Jalife, José, Ward, Peter A.
Formato: Artigo
Idioma:Inglês
Publicado em: 2016
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4988523/
https://ncbi.nlm.nih.gov/pubmed/27521340
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1600091
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