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Defective DNA repair and chromatin organization in patients with quiescent systemic lupus erythematosus
BACKGROUND: Excessive autoantibody production characterizing systemic lupus erythematosus (SLE) occurs irrespective of the disease’s clinical status and is linked to increased lymphocyte apoptosis. Herein, we tested the hypothesis that defective DNA damage repair contributes to increased apoptosis i...
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| Publicado en: | Arthritis Res Ther |
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| Main Authors: | , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado: |
BioMed Central
2016
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| Assuntos: | |
| Acceso en liña: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4973109/ https://ncbi.nlm.nih.gov/pubmed/27492607 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s13075-016-1081-3 |
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