Alpha-1-antitrypsin interacts with gp41 to block HIV-1 entry into CD4+ T lymphocytes

BACKGROUND: Study of a clinic case reveals that alpha-1-antitrypsin (AAT) deficiency is related to CD4+ T cell count decline and AIDS progression, suggesting that AAT might be an endogenous inhibitor of HIV/AIDS. Previous study shows that AAT inhibits HIV-1 replication in infected host cells and the...

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Detaylı Bibliyografya
Yayımlandı:BMC Microbiol
Asıl Yazarlar: Zhou, Xueyuan, Liu, Zhu, Zhang, Jun, Adelsberger, Joseph W., Yang, Jun, Burton, Gregory F.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: BioMed Central 2016
Konular:
Research Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC4966588/
https://ncbi.nlm.nih.gov/pubmed/27473095
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12866-016-0751-2
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