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Hearing, reactive metabolite formation, and oxidative stress in cochleae after a single acute overdose of acetaminophen: an in vivo study
CONTEXT: Although the liver is the primary target organ in acetaminophen (APAP) toxicity, other organs are affected. Previous data suggested chronic APAP abuse can be ototoxic and the mechanism involves APAP-induced oxidative stress and reactive metabolite (NAPQI)-induced endoplasmic reticulum stres...
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| Publicat a: | Toxicol Mech Methods |
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| Autors principals: | , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2016
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4940278/ https://ncbi.nlm.nih.gov/pubmed/26982240 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3109/15376516.2015.1122136 |
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