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Action potential broadening in a presynaptic channelopathy
Brain development and interictal function are unaffected in many paroxysmal neurological channelopathies, possibly explained by homoeostatic plasticity of synaptic transmission. Episodic ataxia type 1 is caused by missense mutations of the potassium channel Kv1.1, which is abundantly expressed in th...
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| Udgivet i: | Nat Commun |
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| Main Authors: | , , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
Nature Publishing Group
2016
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4935806/ https://ncbi.nlm.nih.gov/pubmed/27381274 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/ncomms12102 |
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