Phosphorylation inactivation of endothelial nitric oxide synthesis in pulmonary arterial hypertension

The impairment of vasodilator nitric oxide (NO) production is well accepted as a typical marker of endothelial dysfunction in vascular diseases, including in the pathophysiology of pulmonary arterial hypertension (PAH), but the molecular mechanisms accounting for loss of NO production are unknown. W...

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Bibliografiset tiedot
Julkaisussa:Am J Physiol Lung Cell Mol Physiol
Päätekijät: Ghosh, Sudakshina, Gupta, Manveen, Xu, Weiling, Mavrakis, Deloris A., Janocha, Allison J., Comhair, Suzy A. A., Haque, Mohammad Mahfuzul, Stuehr, Dennis J., Yu, Jun, Polgar, Peter, Naga Prasad, Sathyamangla V., Erzurum, Serpil C.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: American Physiological Society 2016
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Articles
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC4935470/
https://ncbi.nlm.nih.gov/pubmed/27130529
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00092.2016
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