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Mobility and subcellular localization of endogenous, gene-edited Tau differs from that of over-expressed human wild-type and P301L mutant Tau

Alzheimer’s disease (AD) and a subset of frontotemporal dementia termed FTLD-Tau are characterized by a massive, yet incompletely characterized and understood redistribution of Tau. To establish a framework for understanding this pathology, we used the genome-editing tool TALEN and generated Tau-mEO...

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Pubblicato in:Sci Rep
Autori principali: Di Xia, Gutmann, Julia M., Götz, Jürgen
Natura: Artigo
Lingua:Inglês
Pubblicazione: Nature Publishing Group 2016
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC4932628/
https://ncbi.nlm.nih.gov/pubmed/27378256
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/srep29074
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