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Genetic alteration of the metal/redox modulation of Cav3.2 T‐type calcium channel reveals its role in neuronal excitability
KEY POINTS: In this study, we describe a new knock‐in (KI) mouse model that allows the study of the H191‐dependent regulation of T‐type Cav3.2 channels. Sensitivity to zinc, nickel and ascorbate of native Cav3.2 channels is significantly impeded in the dorsal root ganglion (DRG) neurons of this KI m...
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| Veröffentlicht in: | J Physiol |
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| Hauptverfasser: | , , |
| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
John Wiley and Sons Inc.
2016
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4929317/ https://ncbi.nlm.nih.gov/pubmed/26931411 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/JP271925 |
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