UCP2 induces metabolic reprogramming to inhibit proliferation of cancer cells

Invalidation of uncoupling protein 2 (Ucp2) increases glucose utilization and proliferation in normal cells. We recently reported that cancer cells that overexpress UCP2 become less tumorigenic while switching their metabolism from glycolysis to oxidative phosphorylation. UCP2 appears to be a key re...

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Udgivet i:Mol Cell Oncol
Main Authors: Esteves, Pauline, Pecqueur, Claire, Alves-Guerra, Marie-Clotilde
Format: Artigo
Sprog:Inglês
Udgivet: Taylor & Francis 2014
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Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4905249/
https://ncbi.nlm.nih.gov/pubmed/27308391
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4161/23723556.2014.975024
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