Deletion of Interleukin-6 Attenuates Pressure Overload-Induced Left Ventricular Hypertrophy and Dysfunction

RATIONALE: The role of interleukin (IL)-6 in the pathogenesis of cardiac myocyte hypertrophy remains controversial. OBJECTIVE: To conclusively determine whether IL-6 signaling is essential for the development of pressure overload-induced left ventricular (LV) hypertrophy, and to elucidate the underl...

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Bibliografski detalji
Izdano u:Circ Res
Glavni autori: Zhao, Lin, Cheng, Guangming, Jin, Runming, Afzal, Muhammad R., Samanta, Anweshan, Xuan, Yu-Ting, Girgis, Magdy, Elias, Harold K, Zhu, Yanqing, Davani, Arash, Yang, Yanjuan, Chen, Xing, Ye, Sheng, Wang, Ou-Li, Chen, Lei, Hauptman, Jeryl, Vincent, Robert J., Dawn, Buddhadeb
Format: Artigo
Jezik:Inglês
Izdano: 2016
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Article
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC4902783/
https://ncbi.nlm.nih.gov/pubmed/27126808
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCRESAHA.116.308688
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