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Creating a Pro-survival and Anti-inflammatory Phenotype by Modulation of Acetylation in Models of Hemorrhagic and Septic Shock
Shock, regardless of etiology, is characterized by decreased tissue perfusion resulting in cell death, organ dysfunction, and poor survival. Current therapies largely focus on restoring tissue perfusion through resuscitation but have failed to address the specific cellular dysfunction caused by shoc...
Guardat en:
| Publicat a: | Adv Exp Med Biol |
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| Autors principals: | , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2012
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4894314/ https://ncbi.nlm.nih.gov/pubmed/22127890 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/978-1-4419-5638-5_11 |
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