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Complement-mediated ‘bystander’ damage initiates host NLRP3 inflammasome activation
Complement activation has long been associated with inflammation, primarily due to the elaboration of the complement anaphylotoxins C5a and C3a. In this work, we demonstrate that the phagocytosis of complement-opsonized particles promotes host inflammatory responses by a new mechanism that depends o...
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| Publicado no: | J Cell Sci |
|---|---|
| Main Authors: | , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
The Company of Biologists Ltd
2016
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4893798/ https://ncbi.nlm.nih.gov/pubmed/27006116 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1242/jcs.179291 |
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