GITRL modulates the activities of p38 MAPK and STAT3 to promote Th17 cell differentiation in autoimmune arthritis

The glucocorticoid-induced TNFR family-related protein (GITR) and its ligand play a critical role in the pathogenesis of autoimmune arthritis by enhancing the Th17 cell response, but their molecular mechanisms remain largely unclear. This study aims to define the role of p38 mitogen-activated protei...

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Detalhes bibliográficos
Publicado no:Oncotarget
Main Authors: Tang, Xinyi, Tian, Jie, Ma, Jie, Wang, Jiemin, Qi, Chen, Rui, Ke, Wang, Yungang, Xu, Huaxi, Lu, Liwei, Wang, Shengjun
Formato: Artigo
Idioma:Inglês
Publicado em: Impact Journals LLC 2015
Assuntos:
Research Paper: Immunology
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4890989/
https://ncbi.nlm.nih.gov/pubmed/26657118
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.18632/oncotarget.6535
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