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Lysosomal cholesterol accumulation in macrophages leading to coronary atherosclerosis in CD38(−/−) mice
The disruption in transportation of oxLDL‐derived cholesterol and the subsequent lipid accumulation in macrophages are the hallmark events in atherogenesis. Our recent studies demonstrated that lysosomal Ca(2+) messenger of nicotinic acid adenine dinucleotide phosphate (NAADP), an enzymatic product...
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| Vydáno v: | J Cell Mol Med |
|---|---|
| Hlavní autoři: | , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
John Wiley and Sons Inc.
2016
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4882979/ https://ncbi.nlm.nih.gov/pubmed/26818887 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.12788 |
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