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BAFF promotes autoantibody production via TACI-dependent activation of transitional B cells
Mice overexpressing B cell activating factor of the TNF family (BAFF) develop systemic autoimmunity characterized by class-switched anti-nuclear antibodies. Transmembrane activator and CAML interactor (TACI) signals are critical for BAFF-mediated autoimmunity, but the B cell developmental subsets un...
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| 出版年: | J Immunol |
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| 主要な著者: | , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2016
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4868625/ https://ncbi.nlm.nih.gov/pubmed/27022196 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1600017 |
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