טוען...
Retinal glial responses to optic nerve crush are attenuated in Bax-deficient mice and modulated by purinergic signaling pathways
BACKGROUND: Retinal ganglion cell (RGC) soma death is a consequence of optic nerve damage, including in optic neuropathies like glaucoma. The activation of the innate immune network in the retina after nerve damage has been linked to RGC pathology. Since the eye is immune privileged, innate immune f...
שמור ב:
| הוצא לאור ב: | J Neuroinflammation |
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| Main Authors: | , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
BioMed Central
2016
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4850653/ https://ncbi.nlm.nih.gov/pubmed/27126275 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12974-016-0558-y |
| תגים: |
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