Nav1.5 N-terminal domain binding to α1-syntrophin increases membrane density of human Kir2.1, Kir2.2 and Nav1.5 channels

AIMS: Cardiac excitability and refractoriness are largely determined by the function and number of inward rectifier K(+) channels (Kir2.1–2.3), which are differentially expressed in the atria and ventricles, and Nav1.5 channels. We have focused on how Nav1.5 and Kir2.x function within a macromolecul...

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Dettagli Bibliografici
Pubblicato in:Cardiovasc Res
Autori principali: Matamoros, Marcos, Pérez-Hernández, Marta, Guerrero-Serna, Guadalupe, Amorós, Irene, Barana, Adriana, Núñez, Mercedes, Ponce-Balbuena, Daniela, Sacristán, Sandra, Gómez, Ricardo, Tamargo, Juan, Caballero, Ricardo, Jalife, José, Delpón, Eva
Natura: Artigo
Lingua:Inglês
Pubblicazione: Oxford University Press 2016
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ORIGINAL ARTICLES
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC4836625/
https://ncbi.nlm.nih.gov/pubmed/26786162
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/cvr/cvw009
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