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GABAB receptor cell surface export is controlled by an endoplasmic reticulum gatekeeper
Endoplasmic reticulum (ER) release and cell surface export of many G protein-coupled receptors (GPCRs), are tightly regulated. For GABA(B) receptors of GABA, the major mammalian inhibitory neurotransmitter, the ligand-binding GB1 subunit is maintained in the ER by unknown mechanisms in the absence o...
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| Gepubliceerd in: | Mol Psychiatry |
|---|---|
| Hoofdauteurs: | , , , , , , , , , , , , , , , , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
Nature Publishing Group Specialist Journals
2016
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4828513/ https://ncbi.nlm.nih.gov/pubmed/26033241 |
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