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The role of TWEAK/Fn14 signaling in the MPTP-model of Parkinson’s disease

The tumor necrosis factor like weak inducer of apoptosis (TWEAK) and its receptor, fibroblast growth factor-inducible 14 (Fn14), mediate inflammation and neuronal apoptosis in cerebral edema, ischemic stroke and multiple sclerosis. The downstream effectors and pathways linked to TWEAK–Fn14 signaling...

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Detaylı Bibliyografya
Yayımlandı:Neuroscience
Asıl Yazarlar: Mustafa, S., Martin, H.L., Burkly, L., Costa, A., Martins, M.L., Schwaninger, M., Teismann, P.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Elsevier Science 2016
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC4771015/
https://ncbi.nlm.nih.gov/pubmed/26808775
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuroscience.2016.01.034
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