Continuous AMD3100 Treatment Worsens Renal Fibrosis through Regulation of Bone Marrow Derived Pro-Angiogenic Cells Homing and T-Cell-Related Inflammation

AMD3100 is a small molecule inhibitor of chemokine receptor type 4 (CXCR4), which is located in the cell membranes of CD34+ cells and a variety of inflammatory cells and has been reported to reduce organ fibrosis in the lung, liver and myocardium. However, the effect of AMD3100 on renal fibrosis is...

وصف كامل

محفوظ في:
التفاصيل البيبلوغرافية
الحاوية / القاعدة:PLoS One
المؤلفون الرئيسيون: Yang, Juan, Zhu, Fengming, Wang, Xiaohui, Yao, Weiqi, Wang, Meng, Pei, Guangchang, Hu, Zhizhi, Guo, Yujiao, Zhao, Zhi, Wang, Pengge, Mou, Jingyi, Sun, Jie, Zeng, Rui, Xu, Gang, Liao, Wenhui, Yao, Ying
التنسيق: Artigo
اللغة:Inglês
منشور في: Public Library of Science 2016
الموضوعات:
Research Article
الوصول للمادة أونلاين:https://ncbi.nlm.nih.gov/pmc/articles/PMC4763993/
https://ncbi.nlm.nih.gov/pubmed/26900858
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0149926
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