Short peptides from leucyl-tRNA synthetase rescue disease-causing mitochondrial tRNA point mutations

Lignende værker

• The isolated carboxy-terminal domain of human mitochondrial leucyl-tRNA synthetase rescues the pathological phenotype of mitochondrial tRNA mutations in human cells
  af: Perli, Elena, et al.
  Udgivet: (2014)
• The isolated carboxy‐terminal domain of human mitochondrial leucyl‐tRNA synthetase rescues the pathological phenotype of mitochondrial tRNA mutations in human cells
  af: Elena Perli, et al.
  Udgivet: (2014-01-01)
• The phenotypic expression of mitochondrial tRNA-mutations can be modulated by either mitochondrial leucyl-tRNA synthetase or the C-terminal domain thereof
  af: Giordano, Carla, et al.
  Udgivet: (2015)
• Peptide-mimetics derived from leucyl-tRNA synthetase are potential agents for the therapy of mt-tRNA related diseases
  af: Annalinda Pisano, et al.
  Udgivet: (2025-07-01)
• Human mitochondrial leucyl tRNA synthetase can suppress non cognate pathogenic mt-tRNA mutations
  af: Hornig-Do, Hue Tran, et al.
  Udgivet: (2014)