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Erythropoietin Modulates Cerebral and Serum Degradation Products from Excess Calpain Activation following Prenatal Hypoxia-Ischemia

Preterm infants suffer central nervous system (CNS) injury from hypoxia-ischemia and inflammation, termed encephalopathy of prematurity. Mature CNS injury activates caspase and calpain proteases. Erythropoietin (EPO) limits apoptosis mediated by activated caspases, but its role in modulating calpain...

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Bibliografske podrobnosti
izdano v:Dev Neurosci
Main Authors: Jantzie, Lauren L., Winer, Jesse, Corbett, Chris, Robinson, Shenandoah
Format: Artigo
Jezik:Inglês
Izdano: 2015
Teme:
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC4732905/
https://ncbi.nlm.nih.gov/pubmed/26551007
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1159/000441024
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