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Erythropoietin Modulates Cerebral and Serum Degradation Products from Excess Calpain Activation following Prenatal Hypoxia-Ischemia
Preterm infants suffer central nervous system (CNS) injury from hypoxia-ischemia and inflammation, termed encephalopathy of prematurity. Mature CNS injury activates caspase and calpain proteases. Erythropoietin (EPO) limits apoptosis mediated by activated caspases, but its role in modulating calpain...
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| Vydáno v: | Dev Neurosci |
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| Hlavní autoři: | , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
2015
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4732905/ https://ncbi.nlm.nih.gov/pubmed/26551007 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1159/000441024 |
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