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Endoplasmic Reticulum pathology and stress response in neurons precede programmed necrosis after neonatal hypoxia-ischemia

The endoplasmic reticulum (ER) is tasked, among many other functions, with preventing excitotoxicity from killing neurons following neonatal hypoxia-ischemia (HI). With the search for delayed therapies to treat neonatal HI, the study of delayed ER responses becomes relevant. We hypothesized that ER...

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Bibliografiska uppgifter
I publikationen:Int J Dev Neurosci
Huvudupphovsmän: Chavez-Valdez, Raul, Flock, Debbie L., Martin, Lee J., Northington, Frances J.
Materialtyp: Artigo
Språk:Inglês
Publicerad: 2015
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Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC4718855/
https://ncbi.nlm.nih.gov/pubmed/26643212
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ijdevneu.2015.11.007
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