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Endoplasmic Reticulum pathology and stress response in neurons precede programmed necrosis after neonatal hypoxia-ischemia
The endoplasmic reticulum (ER) is tasked, among many other functions, with preventing excitotoxicity from killing neurons following neonatal hypoxia-ischemia (HI). With the search for delayed therapies to treat neonatal HI, the study of delayed ER responses becomes relevant. We hypothesized that ER...
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| Published in: | Int J Dev Neurosci |
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| Main Authors: | , , , |
| Format: | Artigo |
| Language: | Inglês |
| Published: |
2015
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| Subjects: | |
| Online Access: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4718855/ https://ncbi.nlm.nih.gov/pubmed/26643212 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ijdevneu.2015.11.007 |
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