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Acute adriamycin-induced cardiotoxicity is exacerbated by angiotension II
Adriamycin (ADR) increases the production of reactive oxygen species (ROS), which diminishes mitochondrial function. Angiotensin-II stimulates mitochondrial ROS generation. The aim of the study was to examine whether angiotensin converting enzyme (ACE) or renin inhibitors protect against ADR-induced...
Tallennettuna:
| Julkaisussa: | Cytotechnology |
|---|---|
| Päätekijät: | , , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
Springer Netherlands
2014
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4698265/ https://ncbi.nlm.nih.gov/pubmed/25023137 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s10616-014-9748-6 |
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