Acute adriamycin-induced cardiotoxicity is exacerbated by angiotension II

Adriamycin (ADR) increases the production of reactive oxygen species (ROS), which diminishes mitochondrial function. Angiotensin-II stimulates mitochondrial ROS generation. The aim of the study was to examine whether angiotensin converting enzyme (ACE) or renin inhibitors protect against ADR-induced...

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Vydáno v:Cytotechnology
Hlavní autoři: Taskin, Eylem, Kindap, Elvan Kunduz, Ozdogan, Kalender, Aycan, Mukerrem Betul Yerer, Dursun, Nurcan
Médium: Artigo
Jazyk:Inglês
Vydáno: Springer Netherlands 2014
Témata:
Original Research
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC4698265/
https://ncbi.nlm.nih.gov/pubmed/25023137
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s10616-014-9748-6
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