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Human ataxias: a genetic dissection of inositol triphosphate receptor (ITPR1)-dependent signaling
A persistent mystery about the ataxias has been why mutations in genes – many of which are expressed widely in the brain – primarily cause ataxia, and not, for example, epilepsy or dementia. Why should a poly-glutamine stretch in the TATA-binding protein (that is important in all cells) particularly...
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| הוצא לאור ב: | Trends Neurosci |
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| Main Authors: | , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
2010
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4684264/ https://ncbi.nlm.nih.gov/pubmed/20226542 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.tins.2010.02.005 |
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