GSK3β inhibition protects the immature brain from hypoxic-ischemic insult via reduced STAT3 signalling

Hypoxic-ischemic (HI) injury is an important cause of neurological morbidity in neonates. HI leads to pathophysiological responses, including inflammation and oxidative stress that culminate in cell death. Activation of glycogen synthase kinase 3β (GSK3β) and the signal transducer and activator of t...

Descripción completa

Guardado en:
Detalles Bibliográficos
Publicado en:Neuropharmacology
Autores principales: D'Angelo, Barbara, Ek, C. Joakim, Sun, Yanyan, Zhu, Changlian, Sandberg, Mats, Mallard, Carina
Formato: Artigo
Lenguaje:Inglês
Publicado: 2015
Materias:
Article
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC4681657/
https://ncbi.nlm.nih.gov/pubmed/26384655
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuropharm.2015.09.017
Etiquetas: Agregar Etiqueta
Sin Etiquetas, Sea el primero en etiquetar este registro!

Ejemplares similares