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Amyloid Precursor Protein Translation Is Regulated by a 3’UTR Guanine Quadruplex
A central event in Alzheimer’s disease is the accumulation of amyloid β (Aβ) peptides generated by the proteolytic cleavage of the amyloid precursor protein (APP). APP overexpression leads to increased Aβ generation and Alzheimer’s disease in humans and altered neuronal migration and increased long...
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| 出版年: | PLoS One |
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| 主要な著者: | , , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Public Library of Science
2015
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4664259/ https://ncbi.nlm.nih.gov/pubmed/26618502 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0143160 |
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