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Inhibition of K(v) channel expression by NSAIDs depolarizes membrane potential and inhibits cell migration by disrupting calpain signaling
Clinical use of non-steroidal anti-inflammatory drugs (NSAIDs) is well known to cause gastrointestinal ulcer formation via several mechanisms that include inhibiting epithelial cell migration and mucosal restitution. The drug-affected signaling pathways that contribute to inhibition of migration by...
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| Publicado en: | Biochem Pharmacol |
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| Main Authors: | , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado: |
2015
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| Assuntos: | |
| Acceso en liña: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4656099/ https://ncbi.nlm.nih.gov/pubmed/26549367 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bcp.2015.10.017 |
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