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Metabolic vulnerability of KRAS-driven cancer cells

Interfering with anaplerotic utilization of glutamine (Gln) was recently reported to sensitize KRAS-driven cancer cells to the cytotoxic effects of capecitabine and paclitaxel. This effect was due to bypass of a Gln-dependent G(1) cell cycle checkpoint in these cells. This study highlights therapeut...

Ausführliche Beschreibung

Gespeichert in:
Bibliographische Detailangaben
Veröffentlicht in:Mol Cell Oncol
1. Verfasser: Foster, David A
Format: Artigo
Sprache:Inglês
Veröffentlicht: Taylor & Francis 2014
Schlagworte:
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4655600/
https://ncbi.nlm.nih.gov/pubmed/26609545
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4161/23723548.2014.963445
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