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BRAF inhibition generates a host/tumor niche that mediates therapeutic escape

The current study defines a fibroblast-derived niche that facilitates the therapeutic escape of melanoma cells from BRAF inhibition. Vemurafenib treatment led to the release of TGF-β from the melanoma cells that increased the differentiation state of the fibroblasts; an affect associated with fibron...

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書目詳細資料
發表在:J Invest Dermatol
Main Authors: Fedorenko, Inna V., Wargo, Jennifer A., Flaherty, Keith T., Messina, Jane L., Smalley, Keiran S.M.
格式: Artigo
語言:Inglês
出版: 2015
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在線閱讀:https://ncbi.nlm.nih.gov/pmc/articles/PMC4648653/
https://ncbi.nlm.nih.gov/pubmed/26302068
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/jid.2015.329
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