MARCKS Signaling Differentially Regulates Vascular Smooth Muscle and Endothelial Cell Proliferation through a KIS-, p27(kip1)- Dependent Mechanism

BACKGROUND: Overexpression of the myristolated alanine-rich C kinase substrate (MARCKS) occurs in vascular proliferative diseases such as restenosis after bypass surgery. MARCKS knockdown results in arrest of vascular smooth muscle cell (VSMC) proliferation with little effect on endothelial cell (EC...

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Detalhes bibliográficos
Publicado no:PLoS One
Main Authors: Yu, Dan, Makkar, George, Dong, Tuo, Strickland, Dudley K., Sarkar, Rajabrata, Monahan, Thomas Stacey
Formato: Artigo
Idioma:Inglês
Publicado em: Public Library of Science 2015
Assuntos:
Research Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4631550/
https://ncbi.nlm.nih.gov/pubmed/26528715
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0141397
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