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Antenatal endotoxin disrupts lung vitamin D receptor and 25-hydroxyvitamin D 1α-hydroxylase expression in the developing rat

Vitamin D [vit D; 1,25-(OH)(2)D] treatment improves survival and lung alveolar and vascular growth in an experimental model of bronchopulmonary dysplasia (BPD) after antenatal exposure to endotoxin (ETX). However, little is known about lung-specific 1,25-(OH)(2)D(3) regulation during development, es...

पूर्ण विवरण

में बचाया:
ग्रंथसूची विवरण
में प्रकाशित:Am J Physiol Lung Cell Mol Physiol
मुख्य लेखकों: Mandell, Erica, Seedorf, Gregory J., Ryan, Sharon, Gien, Jason, Cramer, Scott D., Abman, Steven H.
स्वरूप: Artigo
भाषा:Inglês
प्रकाशित: American Physiological Society 2015
विषय:
ऑनलाइन पहुंच:https://ncbi.nlm.nih.gov/pmc/articles/PMC4628986/
https://ncbi.nlm.nih.gov/pubmed/26342089
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00253.2015
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