AMPK deficiency in cardiac muscle results in dilated cardiomyopathy in the absence of changes in energy metabolism

AIMS: AMP-activated protein kinase (AMPK) is thought to be a central player in regulating myocardial metabolism and its activation has been shown to inhibit cardiac hypertrophy. Recently, mice with muscle-specific deletion of AMPK β1/β2 subunits (AMPKβ1β2-deficient mice, β1β2M-KO) have been generate...

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Detalhes bibliográficos
Publicado no:Cardiovasc Res
Main Authors: Sung, Miranda M., Zordoky, Beshay N., Bujak, Adam L., Lally, James S.V., Fung, David, Young, Martin E., Horman, Sandrine, Miller, Edward J., Light, Peter E., Kemp, Bruce E., Steinberg, Gregory R., Dyck, Jason R.B.
Formato: Artigo
Idioma:Inglês
Publicado em: Oxford University Press 2015
Assuntos:
Original Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4565988/
https://ncbi.nlm.nih.gov/pubmed/26023060
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/cvr/cvv166
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