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SOD1 misplacing and mitochondrial dysfunction in amyotrophic lateral sclerosis pathogenesis
Amyotrophic lateral sclerosis (ALS) is a fatal motor neuron disease presenting as sporadic (sALS) or familial (fALS) forms. Even if the list of the genes underlining ALS greatly expanded, defects in superoxide dismutase 1 (SOD1), encoding the copper/zinc SOD1, still remain a major cause of fALS and...
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| I publikationen: | Front Cell Neurosci |
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| Huvudupphovsmän: | , , , , |
| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
Frontiers Media S.A.
2015
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4548205/ https://ncbi.nlm.nih.gov/pubmed/26379505 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fncel.2015.00336 |
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