Caveolin-1 deletion exacerbates cardiac interstitial fibrosis by promoting M2 macrophage activation in mice after myocardial infarction

Adverse remodeling following myocardial infarction (MI) leading to heart failure is driven by an imbalanced resolution of inflammation. The macrophage cell is an important control of post-MI inflammation, as macrophage subtypes secrete mediators to either promote inflammation and extend injury (M1 p...

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Detalhes bibliográficos
Publicado no:J Mol Cell Cardiol
Main Authors: Shivshankar, Pooja, Halade, Ganesh V., Calhoun, Cheresa, Escobar, Gladys P., Mehr, Ali J., Jimenez, Fabio, Martinez, Cindy, Bhatnagar, Harshita, Mjaatvedt, Corey H., Lindsey, Merry L., Le Saux, Claude Jourdan
Formato: Artigo
Idioma:Inglês
Publicado em: 2014
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4533121/
https://ncbi.nlm.nih.gov/pubmed/25128086
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2014.07.020
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