Advanced glycation end products induce glomerular sclerosis and albuminuria in normal rats.

High levels of tissue advanced glycation end products (AGEs) that result from the spontaneous modification of proteins by glucose occur in diabetes and aging. To address the potential pathogenic role of AGEs in the glomerulosclerosis of diabetes or nephrosclerosis of aging, doses of AGE-modified rat...

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Detalhes bibliográficos

Publicado no:	Proc Natl Acad Sci U S A
Principais autores:	Vlassara, H, Striker, L J, Teichberg, S, Fuh, H, Li, Y M, Steffes, M
Formato:	Artigo
Idioma:	Inglês
Publicado em:	National Academy of Sciences 1994
Assuntos:	Research Article
Acesso em linha:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC45300/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7972128/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.91.24.11704
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