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Excessive feedback of Cyp26a1 promotes cell non-autonomous loss of retinoic acid signaling
Teratogenic levels of retinoic acid (RA) signaling can cause seemingly contradictory phenotypes indicative of both increases and decreases of RA signaling. However, the mechanisms underlying these contradictory phenotypes are not completely understood. Here, we report that using a hyperactive RA rec...
Tallennettuna:
| Julkaisussa: | Dev Biol |
|---|---|
| Päätekijät: | , , , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
2015
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4529768/ https://ncbi.nlm.nih.gov/pubmed/26116175 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.ydbio.2015.06.008 |
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