Activation of the nimA protein kinase plays a unique role during mitosis that cannot be bypassed by absence of the bimE checkpoint.

Mutation of nimA reversibly arrests cells in late G2 and nimA overexpression promotes premature mitosis. Here we demonstrate that the product of nimA (designated NIMA) has protein kinase activity that can phosphorylate beta-casein but not histone proteins. NIMA kinase activity is cell cycle regulate...

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Dades bibliogràfiques

Publicat a:	EMBO J
Autors principals:	Osmani, A H, O'Donnell, K, Pu, R T, Osmani, S A
Format:	Artigo
Idioma:	Inglês
Publicat:	Nature Publishing Group 1991
Matèries:	Research Article
Accés en línia:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC452969/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/1868838/ https://ncbi.nlm.nih.govhttps://doi.org/10.1002/j.1460-2075.1991.tb07810.x
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