A(3) adenosine receptor agonist prevents the development of paclitaxel-induced neuropathic pain by modulating spinal glial-restricted redox-dependent signaling pathways

Lignende værker

• Spinal neuroimmmune activation is independent of T-cell infiltration and attenuated by A3 adenosine receptor agonists in a model of oxaliplatin-induced peripheral neuropathy
  af: Janes, Kali, et al.
  Udgivet: (2014)
• Chemotherapy-induced pain is promoted by enhanced spinal adenosine kinase levels via astrocyte-dependent mechanisms
  af: Wahlman, Carrie, et al.
  Udgivet: (2018)
• Bioenergetic deficits in peripheral nerve sensory axons during chemotherapy-induced neuropathic pain resulting from peroxynitrite-mediated post-translational nitration of mitochondrial superoxide dismutase
  af: Janes, Kali, et al.
  Udgivet: (2013)
• Targeting the Overproduction of Peroxynitrite for the Prevention and Reversal of Paclitaxel-Induced Neuropathic Pain
  af: Doyle, Timothy, et al.
  Udgivet: (2012)
• Highly selective A3 adenosine receptor agonists relieve chronic neuropathic pain
  af: Salvemini, Daniela, et al.
  Udgivet: (2017)